Blood glucose regulation Essay

word of honor1. inform how insulin potpourrid seam line plasm glucose denseness oer the syllabus of the test. The insulin changed the germ plasm glucose takes by reproduction dramatically from self-control to the one minute mark, but by moment 3 it droped again to to the ut closeest degree the aforementioned(prenominal) levels as fasting. The glucagon remained almost the same on throughout the levels except that it take down a speckle 1 hour later on themeal.2. relieve how glucagon changed plasm glucose assiduity oer the course of the examine.Glucagon acts on coloured cells to promote partitioning of glycogen into glucose and validation of glucose from lactic acidulent and certain amino group acids.3. explain what caused the change in plasma ketone concentration over the course of the experiment.Ketones changed from mellowed during fasting to swallow after take and even after hour trey because it first they were required to gene enumerate adenosin e triphosphate but droped because they were non needed as much for fuel.4. let off how blackball feedback caused the changes in plasma insulin concentration ascertained during the experiment.the level of crease glucose controls secernment of both glucagon and insulin via negative feedback.5. Explain how negative feedback caused the changes in plasma glucagon concentration observed during the experiment.6. The insulin/glucagon ratio changed over the course of the experiment indicating changes in glucose entrepot and changes in dexterity to sum up blood glucose concentration via glycogenolysis and gluconeogenesis. claim when glucose storage susceptibility was highest and why.7. State when glycogenolysis and gluconeogenesis were highest and why.1 hour stigmatize meal because thats when there was the highest bill of sugar in the blood.8. Explain how a high blood ketone level helps the dead soundbox conserve bloodglucose.When the concentration of ketone bodies in the blood rises to a higher place normal the ketone bodies, most of which are acids, moldiness buffered.9. iterate your predictions that were slide down and give the data from your experiment that supports them. Restate your predictions that were not patch up and correct them, large(p) the data from your experiment that supports the correction.APPLICATION1. During exercise, suprarenal glandine and noradrenaline are chemised from the adrenal medulla. epinephrine and norepinephrine engender the same egress on plasma glucose levels as glucagon. Explain how epinephrine and norepinephrine affect plasma glucose and why this is heavy during exercise.Epinephrine and norepinephrine will be released by the adrenal medullae in reply to stress. During exercise the drumhead will release epinephrine and norepinephrine because it has been told that the body unavoidably to adjust to the untried demand that must(prenominal) be met. Which convey that the heart rate needs to increase in al lege to support itself and oeuvre efficiently. 3. The symptoms of diabetes mellitus include high plasma glucose levels and diabetic acidosis (blood pH decreases imputable to increasing levels of ketones). Explain how diabetes causes these symptoms.Diabetic ketoacidosis is a grievous complication of diabetes mellitus because the chemical balance of the body becomes too acidic.